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Cell Marque fli1 clone mrq-1 antibody
Fli1 Clone Mrq 1 Antibody, supplied by Cell Marque, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fli1+clone+mrq-1+antibody/fli+1+antibody/pm40278957-16-72-77
Average 90 stars, based on 1 article reviews
fli1 clone mrq-1 antibody - by Bioz Stars, 2026-11
90/100 stars

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Article Title: Melanotic Neuroectodermal Tumor of Infancy: Clinicopathological Evaluation of a 10-Year Consecutive Case Series from a Tertiary Cancer Center.
Article Snippet: Melanotic Neuroectodermal Tumor of Infancy (MNTI) is a rare, locally aggressive tumor consisting of a characteristic biphasic population of small neuroblast-like cells and larger melanin-containing epithelioid cells [1].. It is recognized now as a locally aggressive and rarely metastasizing entity [1–2].. Most cases of MNTI described to date have been reported in the head and neck region of infants where they can mimic a wide range of tumors [3].



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Cell Marque fli1 clone mrq-1 antibody
Fli1 Clone Mrq 1 Antibody, supplied by Cell Marque, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fli1+clone+mrq-1+antibody/fli+1+antibody/pm40278957-16-72-77
Average 90 stars, based on 1 article reviews
fli1 clone mrq-1 antibody - by Bioz Stars, 2026-11
90/100 stars
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Cell Marque mouse monoclonal antibody to human fli1 clone mrq-1
Antral glands (A) , oxyntic glands (B) , IM (C) , representative tumor with high <t>FLI1</t> H-score of 199 (D–F) , representative tumor with low FLI1 H-score of 88 (G–I) . E, F, H and I show tumor samples with nuclear FLI1 IHC staining, at low (E and H) and high magnification (F and I). (I) Arrows point to tumor nuclei with little to no FLI1 positivity. Arrowhead points to stromal cell nucleus with strong FLI1 positivity, serving as internal positive control. A-D and G are H&E stains. Low power H&E (J) of stomach with high-grade dysplasia (K) and invasive adenocarcinoma (L) . High power view of K and L inset are shown as K * and L * . Panel M shows FLI1 IHC of the case represented in panel J. High-grade dysplasia with preserved nuclear FLI1 expression is shown in the inset N and as high power view in N * . Invasive adenocarcinoma with loss of nuclear FLI1 expression is shown in the inset O and as high power view in O * , where the arrows indicate the nuclei of invasive adenocarcinoma. Original magnification 200×.
Mouse Monoclonal Antibody To Human Fli1 Clone Mrq 1, supplied by Cell Marque, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fli1+clone+mrq-1+antibody/fli+1+antibody/pmc06570468-157-12-16
Average 90 stars, based on 1 article reviews
mouse monoclonal antibody to human fli1 clone mrq-1 - by Bioz Stars, 2026-11
90/100 stars
  Buy from Supplier

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Antral glands (A) , oxyntic glands (B) , IM (C) , representative tumor with high FLI1 H-score of 199 (D–F) , representative tumor with low FLI1 H-score of 88 (G–I) . E, F, H and I show tumor samples with nuclear FLI1 IHC staining, at low (E and H) and high magnification (F and I). (I) Arrows point to tumor nuclei with little to no FLI1 positivity. Arrowhead points to stromal cell nucleus with strong FLI1 positivity, serving as internal positive control. A-D and G are H&E stains. Low power H&E (J) of stomach with high-grade dysplasia (K) and invasive adenocarcinoma (L) . High power view of K and L inset are shown as K * and L * . Panel M shows FLI1 IHC of the case represented in panel J. High-grade dysplasia with preserved nuclear FLI1 expression is shown in the inset N and as high power view in N * . Invasive adenocarcinoma with loss of nuclear FLI1 expression is shown in the inset O and as high power view in O * , where the arrows indicate the nuclei of invasive adenocarcinoma. Original magnification 200×.

Journal: Oncotarget

Article Title: Downregulation of Friend Leukemia Integration 1 ( FLI1 ) follows the stepwise progression to gastric adenocarcinoma

doi: 10.18632/oncotarget.26974

Figure Lengend Snippet: Antral glands (A) , oxyntic glands (B) , IM (C) , representative tumor with high FLI1 H-score of 199 (D–F) , representative tumor with low FLI1 H-score of 88 (G–I) . E, F, H and I show tumor samples with nuclear FLI1 IHC staining, at low (E and H) and high magnification (F and I). (I) Arrows point to tumor nuclei with little to no FLI1 positivity. Arrowhead points to stromal cell nucleus with strong FLI1 positivity, serving as internal positive control. A-D and G are H&E stains. Low power H&E (J) of stomach with high-grade dysplasia (K) and invasive adenocarcinoma (L) . High power view of K and L inset are shown as K * and L * . Panel M shows FLI1 IHC of the case represented in panel J. High-grade dysplasia with preserved nuclear FLI1 expression is shown in the inset N and as high power view in N * . Invasive adenocarcinoma with loss of nuclear FLI1 expression is shown in the inset O and as high power view in O * , where the arrows indicate the nuclei of invasive adenocarcinoma. Original magnification 200×.

Article Snippet: Tissue sections were stained using a clinically validated mouse monoclonal antibody to human FLI1 (clone MRQ-1, Cell Marque).

Techniques: Immunohistochemistry, Positive Control, Expressing

Bean plots showing the distribution of FLI1 H-scores for normal, intestinal metaplasia (IM), low-grade dysplasia (LGD), high-grade dysplasia (HGD), and gastric adenocarcinoma (GC), with mean H-scores (represented by the largest line in each bean plot) of 187.9 (SEM = 3.25), 193.7 (SEM = 2.37), 172.3 (SEM = 15.2), 140.6 (SEM = 14.8), and 108.2 (SEM = 14.6), respectively. A P value <0.05 was considered statistically significant. Significant differences in FLI1 H-scores were detected for normal vs GC (P = 1.2×10 -5 ), IM vs GC (P = 1.4×10 -6 ), and LGD vs GC (P = 0.006).

Journal: Oncotarget

Article Title: Downregulation of Friend Leukemia Integration 1 ( FLI1 ) follows the stepwise progression to gastric adenocarcinoma

doi: 10.18632/oncotarget.26974

Figure Lengend Snippet: Bean plots showing the distribution of FLI1 H-scores for normal, intestinal metaplasia (IM), low-grade dysplasia (LGD), high-grade dysplasia (HGD), and gastric adenocarcinoma (GC), with mean H-scores (represented by the largest line in each bean plot) of 187.9 (SEM = 3.25), 193.7 (SEM = 2.37), 172.3 (SEM = 15.2), 140.6 (SEM = 14.8), and 108.2 (SEM = 14.6), respectively. A P value <0.05 was considered statistically significant. Significant differences in FLI1 H-scores were detected for normal vs GC (P = 1.2×10 -5 ), IM vs GC (P = 1.4×10 -6 ), and LGD vs GC (P = 0.006).

Article Snippet: Tissue sections were stained using a clinically validated mouse monoclonal antibody to human FLI1 (clone MRQ-1, Cell Marque).

Techniques:

Survival plots displaying survival probability (y-axis) vs time in days (x-axis). (A) Survival plot for all patients analyzed in the TMAs with FLI1 H-score cutoff of 100 to separate high and low FLI1 -expressing tumors. (B) Survival plot for all patients with well and moderately differentiated tumors, using a FLI1 H-score cutoff of 100 to separate high and low FLI1 -expressing tumors. (C) Survival plot for all patients with poorly differentiated tumors, using a FLI1 H-score cutoff of 110.5 to separate high and low FLI1 -expressing tumors. The Cox proportional hazards (PH) model and the Mantel-Haenszel log-rank P values are displayed. A P value <0.05 was considered statistically significant.

Journal: Oncotarget

Article Title: Downregulation of Friend Leukemia Integration 1 ( FLI1 ) follows the stepwise progression to gastric adenocarcinoma

doi: 10.18632/oncotarget.26974

Figure Lengend Snippet: Survival plots displaying survival probability (y-axis) vs time in days (x-axis). (A) Survival plot for all patients analyzed in the TMAs with FLI1 H-score cutoff of 100 to separate high and low FLI1 -expressing tumors. (B) Survival plot for all patients with well and moderately differentiated tumors, using a FLI1 H-score cutoff of 100 to separate high and low FLI1 -expressing tumors. (C) Survival plot for all patients with poorly differentiated tumors, using a FLI1 H-score cutoff of 110.5 to separate high and low FLI1 -expressing tumors. The Cox proportional hazards (PH) model and the Mantel-Haenszel log-rank P values are displayed. A P value <0.05 was considered statistically significant.

Article Snippet: Tissue sections were stained using a clinically validated mouse monoclonal antibody to human FLI1 (clone MRQ-1, Cell Marque).

Techniques: Expressing

(A) Normalized (log2) expression (Fragments per Kb of transcript per Million fragments mapped, FPKM) of FLI1 in gastric adenocarcinoma cell lines (FU97, NUGC3, AGS, MKN74, SNU638). (B) Western blots performed with anti-FLI1 (top, ~55kDa) and anti-β-actin (bottom, ~42kDa) antibodies on protein extracts from human gastric adenocarcinoma cell lines. Invasion assay (C) and proliferation assay (D) of AGS cells transduced with either control or FLI1 expression vector lentiviral particles.

Journal: Oncotarget

Article Title: Downregulation of Friend Leukemia Integration 1 ( FLI1 ) follows the stepwise progression to gastric adenocarcinoma

doi: 10.18632/oncotarget.26974

Figure Lengend Snippet: (A) Normalized (log2) expression (Fragments per Kb of transcript per Million fragments mapped, FPKM) of FLI1 in gastric adenocarcinoma cell lines (FU97, NUGC3, AGS, MKN74, SNU638). (B) Western blots performed with anti-FLI1 (top, ~55kDa) and anti-β-actin (bottom, ~42kDa) antibodies on protein extracts from human gastric adenocarcinoma cell lines. Invasion assay (C) and proliferation assay (D) of AGS cells transduced with either control or FLI1 expression vector lentiviral particles.

Article Snippet: Tissue sections were stained using a clinically validated mouse monoclonal antibody to human FLI1 (clone MRQ-1, Cell Marque).

Techniques: Expressing, Western Blot, Invasion Assay, Proliferation Assay, Transduction, Control, Plasmid Preparation